“From about the 1960s, it became widely known that exposure to asbestos dust and fibres could give rise not only to asbestosis and other pulmonary diseases, but also to the risk of developing a mesothelioma. This is a malignant tumour, usually of the pleura, sometimes of the peritoneum. … It is a condition which may be latent for many years, usually for 30-40 years or more; development of the condition may take as short a period as ten years, but it is thought that this is the period which elapses between the mutation of the first cell and the manifestation of symptoms of the condition. It is invariably fatal, and death usually occurs within one to two years of the condition being diagnosed. … It is not known what level of exposure to asbestos dust and fibre can be tolerated without significant risk of developing a mesothelioma, but it is known that those living in urban environments (although without occupational exposure) inhale large numbers of asbestos fibres without developing a mesothelioma. It is accepted that the risk of developing a mesothelioma increases in proportion to the quantity of asbestos dust and fibres inhaled: the greater the quantity of dust and fibre inhaled, the greater the risk. But the condition may be caused by a single fibre, or a few fibres, or many fibres: medical opinion holds none of these possibilities to be more probably than any other, and the condition once caused is not aggravated by further exposure. … There is no way of identifying, even on a balance of probabilities, the source of the fibre or fibres which initiated the genetic process which culminated in the malignant tumour.”
“whether, in the special circumstances of such a case, principle, authority or policy requires or justifies a modified approach to proof of causation.”
“when a victim contracts mesothelioma each person who has, in breach of duty, been responsible for exposing the victim to a significant quantity of asbestos dust and thus creating a `material increase in risk` of the victim contracting the disease will be held to be jointly and severally liable for causing the disease.”
“107. Liability for mesothelioma falls on anyone who has materially increased the risk of the victim contracting the disease. What constitutes a material increase of risk? The parties were, I think, agreed that the insertion of the word “material” is intended to exclude an increase of risk that is so insignificant that the court will properly disregard it on the de minimis principle. Mr Stuart-Smith submitted that there should be a test of what is de minimis, or immaterial, which can be applied in all cases. Exposure should be held immaterial if it did not at least double the environmental exposure to which the victim was subject. It does not seem to me that there is any justification for adopting the “doubles the risk” test as the bench mark of what constitutes a material increase in risk. Indeed, if one were to accept Mr Stuart-Smith`s argument that the “doubles the risk” test establishes causation, his de minimis argument would amount to saying that no exposure is material for the purpose of the Fairchild/Barker test unless on balance of probability it was causative of the mesothelioma. That cannot be right. 108. I doubt whether it is ever possible to define, in quantitative terms, what for the purposes of the application of any principle of law is de minimis. This must be a question for the judge on the facts of the particular case. In the case of mesothelioma, a stage must be reached at which, even allowing for the possibility that exposure to asbestos can have a cumulative effect, a particular exposure is too insignificant to be taken into account, having regard to the overall exposure that has taken place.”
“111. The reality is that in the current state of knowledge about the disease, the only circumstances in which a court will be able to conclude that wrongful exposure of a mesothelioma victim to asbestos dust did not materially increase the victim`s risk of contracting the disease will be where the exposure was insignificant compared to the exposure from other sources.”
“10. … I recall being told that workmen were going to come into the offices to remove the old partitions and replace them over the course of the weekend. I recall coming back to work on the Monday after the work had been completed and there were no partitions between the offices. This is because it took a week for the new partitions to be put in. I believe that the partitions and area around them contained asbestos. I remember being notified of the asbestos removal within the offices when I was working at Freemans. At the time I took no notice of it as I did not know that asbestos was dangerous. 11. I do recall coming into the office on the Monday after the work had been completed and there being quite a lot of dust on the desks and around the floor. Again, I thought nothing of it at the time. During the course of that week cleaners would have come in to clean the offices as they did every week.”
“I was exposed to asbestos when working in various locations including warehouses. The warehouses may have contained asbestos and were dusty. In one of the offices I worked there was refurbishment work going on where partitions were put up and old partitions were removed. This was dusty work.”
“Q. Do you remember there being any dust when the other panels were put back in again and fixed to …? A. No, not particularly, I can`t remember. Q. Because is it not possible that what you are recalling in terms of Dennis`s reaction was when the panels were put back in again? A. It could be. I could not honestly say. I do remember him saying ‘I can taste the dust in my mouth.’ Q. Similarly, I will suggest to you that it is possible, given the passage of time, that the memo you are referring to either referred to other works or the dust that you saw at the time may have been either it being put back in again or non-asbestos dust such as taking out the partitions in the middle which you do not remember but Dennis does? A. The memo definitely related to the removal of the asbestos and I do remember Dennis saying, ‘I can taste dust in my mouth’. Q. What I am suggesting is that you may be right that there was a memo about asbestos, it may have been on a different occasion and it may be that any dust that was left was actually other dust that was not asbestos dust because you would not be able to tell the difference? A. I wouldn’t tell the difference, no. I mean, I wasn’t in his office. The memo definitely related to the removal of the panels with the asbestos. They wouldn’t tell us that they were going to remove them and then tell us that they were going to replace them with something else after the event. …”
“Q. … You were asked a lot of questions about the sequencing when you saw this dust and when the memo was delivered. What is your best recollection about seeing the dust?”
“166. It is important that judges should bear in mind that the Fairchild exception itself represents what the House of Lords considered to be the proper balance between the interests of claimants and defendants in these cases. Especially, having regard to the harrowing nature of the illness, judges, both at first instance and on appeal, must resist any temptation to give the claimant`s case an additional boost by taking a lax approach to the proof of the essential elements. That could only result in the balance struck by the Fairchild exception being distorted.”
“15. An obvious difficulty which affects allegations and oral evidence based on recollection of events which occurred several years ago is the unreliability of human memory. 16. While everyone knows that memory is fallible, I do not believe that the legal system has sufficiently absorbed the lessons of a century of psychological research into the nature of memory and the unreliability of eyewitness testimony. One of the most important lessons of such research is that in everyday life we are not aware of the extent to which our own and other people`s memories are unreliable and believe our memories to be more faithful than they are. Two common (and related) errors are to suppose: (1) that the stronger and more vivid is our feeling or experience of recollection, the more likely the recollection is to be accurate; and (2) that the more confident another person is in their recollection, the more likely their recollection is to be accurate. … 19. The process of civil litigation itself subjects the memories of witnesses to powerful biases. The nature of litigation is such that witnesses often have a stake in a particular version of events. This is obvious where the witness is a party or has a tie of loyalty (such as an employment relationship) to a party to the proceedings. Other, more subtle influences include allegiances created by the process of preparing a witness statement and of coming to court to give evidence for one side in the dispute. A desire to assist, or least not to prejudice, the party who has called the witness or that party`s lawyers, as well as a natural desire to give a good impression in a public forum, can be significant motivating forces. 20. Considerable interference with memory is also introduced in civil litigation by the procedure of preparing for trial. A witness is asked to make a statement, often (as in the present case) when a long time has already elapsed since the relevant events. The statement is usually drafted for the witness by a lawyer who is inevitably conscious of the significance for the issues in the case of what the witness does nor does not say. …The statement may go through several iterations before it is finalized. Then, usually months later, the witness will be asked to re-read his or her statement and review documents again before giving evidence in court. The effect of this process is to establish in the mind of the witness the matters recorded in his or her statement and the other written material, whether they be true or false, and to cause the witness`s memory of events to be based increasingly on this material and later interpretations of it rather than on the original experience of the events. … 22. … This does not mean that oral testimony serves no useful purpose … But its value lies largely, as I see it, in the opportunity which cross-examination affords to subject the documentary record to critical scrutiny and to gauge the personality, motivations and working practices of a witness, rather than in testimony of what the witness recalls of particular conversations and events. Above all, it is important to avoid the fallacy of supposing that, because a witness has confidence in his or her recollection and is honest, evidence based on that recollection provides any reliable guide to the truth.”
“42. The process of attempting to remember events in the distant past is an inherently fallible one; it is a process that is highly susceptible to error and inaccuracy. Our efforts to think back many years to recollect the details of past events are liable to be affected by numerous external influences; involvement in civil litigation can in itself operate as a significant influence. All remembering of events many years ago involves processes of a reconstructive nature; these processes are largely unconscious with a result, as Leggatt J said, that the strength, vividness and apparent authenticity of memories are often not reliable markers of their truth. Having seen and heard the pursuer give evidence, I have come to the view that I must evaluate the reliability of his claimed recollections with caution. I have, wherever possible, tested his evidence against other evidence in the case and I have considered objectively where the probabilities lie.”
“64. These latter observations [referring to para 19 in Gestmin] are, in my view, particularly apposite in the present case in relation to the evidence of the witnesses called by the claimant. The tragic nature of the case and the natural desire to assist in any proper way, are inevitable human reactions. These, and the other factors referred to in the passages quoted above, make it all the more important to test the recollection of the witnesses against contemporaneous documentation.”
“ The risk of asbestos associated disease is related to cumulative exposure - often referred to as the dose received. This is the product of the average airborne fibre count (measured in fibres/ml of air breathed) encountered at work, and the duration of exposure. Duration is expressed in years (a working year usually being taken as 1920 hours) and the resulting unit of cumulative exposure is the fibre/ml year. Thus, a cumulative exposure of 10 fibre/ml years would result from ten years working in an average concentration of 1 fibre/ml, or 2 years in 5 fibres/ml … and so on.”
“15. For the illustrative purpose of providing an estimate of exposure we have each, based on [our] own training and experience, referred to published examples of measured asbestos fibre concentrations, should the Court find that asbestos infill panels were removed without precautions with, as a result, asbestos-containing dust present to surfaces on the Monday. We agree that the extent to which asbestos was present in the dust, if at all, would have depended upon the extent of the asbestos infill panels, how they were removed, the extent of any non-asbestos materials removed/disturbed, and the extent to which any cleaning/removal of waste took place. Nevertheless, we agree that whilst such matters are not known and that we have cited different studies, the levels of exposure referred to by each of us are not too dissimilar, as follows: Mr Raper exposure ranging from around 0.04 fibres/ml or 0.05 fibres/ml to 0.29 fibres/ml during the initial disturbance of deposited material (source: IOM, 2009) Mr Stear exposure ranging from around 0.01 fibres/ml to 0.1 fibres/ml during the initial disturbance of deposited materials (source: Cherrie & Cowie, 2004 and Bailey et al, 1988) We have each adopted a convention that exposure for the remainder of the week would have occurred at 10% of exposure experienced during the disturbance of deposited materials; we agree that the actual decay in airborne asbestos fibre concentrations may not be a linear relationship and the approach we have applied has been taken to provide an illustration of decay in asbestos fibre concentrations.”
“16. We also agree that the magnitude of the Deceased`s asbestos dose will depend on findings of the Court with regard to whether [exposure] occurred and, if it did, the frequency, duration and extent to which the Deceased was exposed. Nevertheless, we have provided different estimates of the Deceased’s cumulative asbestos dose as follows: Mr Raper approximately 0.0010 fibre/ml years Mr Stear 0.00021 to 0.00058 fibre/ml years (midpoint of about 0.0004 fibre/ml years).”
“17. We agree that neither of these dose estimates should be taken as definitive and should only be considered illustrative of the likely magnitude of dose should the Court find that exposure occurred. We agree that whether such low dose was materially causative is a matter for medical comment and ultimately for the Court. Whilst we both agree that the Deceased`s exposure to asbestos would have been very low, we agree that such exposure would have been to a level in excess of published background levels; Mr Raper further considers instantaneous exposure such as that presented in his report and at 15(a) above would have been at some 80 to 290,000 times above such published background concentrations.”
“ 21. … For the claim to succeed, the judge needed to be satisfied that the extent and duration of the exposure had constituted a material increase in the risk to the Deceased of contracting mesothelioma. No specific measurement of the duration is necessary and the Recorder was right to resist the invitation to fix one.”
“ 44. …a judge must determine the degree of exposure to asbestos fibres to which Mr Williams was actually subjected and whether that was a de minimis or a material exposure. If it was a de minimis exposure then there could be no question of a breach of duty, as the judge recognised. But assuming that the exposure was more than de minimis, it was, in my view, necessary to ask a further question. That is whether, given the degree of actual exposure, it ought to have been reasonably foreseeable to the University (with the knowledge a reasonable University should have had in 1974) that, as a result, Mr Williams would have been likely to be exposed to the risk of personal injury in the form of contracting mesothelioma. To determine that question, it seems to me the judge had to make findings about (1) the actual level of exposure to asbestos fibre that Mr Williams was exposed …”
“So long as it was not insignificant, each employer`s wrongful exposure of its employee to asbestos dust, and, hence, to the risk of contracting mesothelioma, should be regarded by the law as a sufficient degree of causal connection.”
“I suggest that a reasonable approach to determining whether a risk from an identified exposure was more than de minimis would be to consider whether the exposure was negligible, ie something so small that it is properly capable of being neglected. I suggest that a dose of asbestos which is properly capable of being neglected could be defined as a dose which a reasonable person who is well informed about the relation between asbestos and mesothelioma would not be worried to discover he had inhaled.”
“ a dose of asbestos which was properly capable of being neglected could be defined as a dose which a medical practitioner who is aware of the medical risks would define as something that the average patient should not worry about.”
“If the court were to accept the [Deceased’s] evidence, on the basis of either Mr Raper’s opinion or Mr Stear`s opinion he sustained more than usual background exposure to amosite asbestos over a period of at least several days. Amosite is a type of amphibole with high potency to cause mesothelioma. Asbestos exposure increases the risk of mesothelioma with no evidence for a threshold dose below which there is no risk. The combination of uncertainties inherent in the scientific model used to establish risks at low levels of exposure, inability to account for individual susceptibility, and uncertainties inherent in estimating the dose which [the Deceased] received means the calculation of the resultant risk in numerical terms using the Hodgson and Darnton tables confers spurious precision on what is at best an exercise in approximation with very wide confidence levels. For example, if the upper end of the range quoted by Hodgson and Darnton were correct ie 15 rather than the 2 per 100,000 quoted by Dr Moore-Gillon, and Mr Raper’s estimate of 1.7 times the upper end of Mr Stear’s estimated range were to be preferred, the estimated risk would have been 7.5 x 1.7, ie 12.75 times higher than that quoted by Dr Moore-Gillon ie 1.275 per 100,000 which is more than the upper boundary of Hodgson and Darnton’s ‘insignificant’ or ‘broadly acceptable’ range of up to 1 in 100,000. In my opinion it is safe to conclude that the exposure alleged will have given rise to a very small but more than negligible, ie properly capable of being neglected, risk of mesothelioma, but the evidence is inadequate to justify precise conclusions regarding the size of the risk.”
‘one has to look at all the facts of the case and try and come to a sense of whether this is a significant exposure or not.’
“it is all we have but we should use it appropriately. It was designed for regulatory purposes and it gives us an approximation of the risk. But, in my view, it should not be used to calculate an exact figure for the risk for an individual from an identified exposure with all the caveats about the degree of uncertainly in this calculation.” iv) agreed arithmetically with Dr Moore-Gillon`s calculation of the risk but was unable to offer any competing figure. v) conceded that he was unable to say at what risk level he would conclude that the risk in this case was not material. This was very much like his approach in the Phurnacite litigation in relation to asbestos-induced lung cancer that any material exposure should be viewed as being contributory towards disease, which approach was rejected by Swift J when she said: “8.22. Dr Rudd said that, in order to assess of the contribution made by a carcinogen, it is necessary to refer to the epidemiological evidence and to look at risk factors. He made clear that the issue of the magnitude of the increase in risk that should be considered sufficient to establish whether the contribution made by the carcinogen had been `material` was a matter for the court. From the medical point of view, even very light exposure might contribute to the carcinogenic process. However, a court might consider that the exposure had been so light that it should not attract an award of damages. He did not offer an opinion as to the size of risk that should be accepted by the court as constituting a `material` contribution for the purposes of establishing legal causation … 8.60. Dr Rudd suggested that it would be necessary for the court to examine the risk factors and to make a judgment as to where the line of materiality should be drawn, according to the court`s view as to whether it would be reasonable to compensate an individual for a specific level of increased risk. … Such an exercise would involve an arbitrary decision on my part as to where to draw the line. … It does not seem to me that it would be permissible for me to carry out such an exercise.”
“ I am not sure that I would even say that it is not one I share. I would not want to give the wrong impression. That is a very small risk. I would agree with him entirely that it is a very small risk and I would reassure a patient that it was a very small risk, but whether that equates to a decision of the court as to whether it is a risk which should be regarded as culpable is a different question.”
“65. … Mr Feeny`s argument is that the proportion of exposure to asbestos fibres that Mr Williams suffered when working [in the Defendant`s employment] was, by comparison with the exposure that he must have suffered elsewhere, a de minimis proportion, which meant that it would be possible to find that [such] exposure … had caused a material increase in the risk of Mr Williams contracting mesothelioma. … 72. However, as Lord Phillips also stated, the question of whether the tortious exposure to asbestos fibres was material is a question of fact for the judge in each case. The judge might or might not be assisted by mathematical comparisons with other real or hypothetical exposure situations as was attempted at the trial in this case. The judge might be assisted by epidemiological evidence. But, as I read the judgments in [Sienkiewicz v Greif], all the judge has to do, ultimately, is to make a finding of fact that the tortious exposure of the victim to asbestos at the hands of the defendant materially increased the risk that the victim would contract mesothelioma. He does not have to do a comparative exercise. So I would reject Mr Feeny`s primary submission on the causation issue.”
“64. … In order for the pursuer to succeed in establishing negligence against the defenders he must establish, on the balance of probabilities, the actual level of asbestos dust to which he was exposed. It is essential for the actual level of exposure to be proved because otherwise the court cannot decide whether the exposure was more than de minimis, in which case liability could not arise. In Sienkiewicz … the Supreme Court held that the claimant must prove that the acts or omissions of a particular defendant had materially increased the risk of contracting mesothelioma. The Supreme Court went on to explain that a material increase means a degree of risk that is more than minimal; it is for the trial judge to determine, on the facts of each case, whether the increase in risk was minimal in this sense.”