“The diagnosis not being in dispute, Dr Warburton’s reports also need not be considered”
“The claimant’s pleaded case does not allege that he was exposed to dust while cutting as opposed to merely handling, asbestos cement. His first statement similarly refers only to cutting millboard. Mr Stear estimates the exposure from cement at 0.021 to 0.029 fibres/ ml years. Such exposure, an order of magnitude less than either expert’s estimate of exposure from cutting millboard, is de minimis.”
“If the millboard did not contain asbestos, exposure from cement was minimal. Even if it did however, guidance at the time did not mandate precautions where exposure was limited and sporadic.”
“In my view therefore on the balance of probabilities if the court accepts that Mr Harrison has had sufficient asbestos exposure to meet or exceed the Helsinki criteria then in my view he has had asbestosis for a number of years.... Mesothelioma is a rare tumour in persons who have not been exposed to asbestos occurring with an annual incidence of around 1 per million population and most cases occur in persons who have been so exposed. Occasional spontaneous cases unrelated to asbestos exposure do occur however, when there is a history of past asbestos exposure the balance of probabilities strongly favours that exposure having been responsible for a mesothelioma which occurs subsequently.”
“Mesothelioma can occur after low level asbestos exposure and there is no threshold dose of asbestos below which there is no risk. However, the risk that mesothelioma will occur increases in proportion to the dose of asbestos received and successive periods of exposure each augment the risk that mesothelioma will occur”and “in my view on the balance of probabilities Mr Harrison has developed an asbestos related plural mesothelioma as a consequence of his previous occupational asbestos exposure.”
“…Mr Harrison worked for several employers on projects requiring general joinery mostly shuttering work for concrete which did not involve asbestos exposure.”
“Mr Harrison recalled using millboard at times. He remembered cutting it with a handsaw and installing it under windowsills in domestic properties at the Guardian Insurance building.”
“Whilst work with asbestos cement does not generate highly elevated airborne fibre concentrations, I have carried out airborne monitoring on activities resulting in airborne fibre concentrations in excess of 0.1 f/mls.”
“Mr Harrison's exposure to asbestos is stated to arise from use of asbestos insulating board, being in close proximity to the mixing and installation of asbestos pipe insulation, drilling of asbestos insulation blocks and being in close proximity to the disturbance of asbestos sprayed coating in addition to the use of other, less friable asbestos products such as asbestos paper and asbestos cement.”
“Mr Harrison worked for this employer as one of four in the tax year 1966/67 and one of six in the tax year 1967/68. He says that he worked on a further contract for the construction of the Guardian insurance building in Blackpool, now the AXA building. His primary role was joinery for concrete shuttering again, but he also fitted panels beneath windows (transom panels) using asbestos millboard, which he describes as a ‘brownie colour’ and asbestos sheeting as another layer which he says was grey. Transom panels are usually made from asbestos cement on the outer side as it is largely weatherproof, especially when painted, therefore I find it reasonable this is what Mr Harrison meant by his description of asbestos sheeting. The millboard layer would have been for the purpose of additional insulation or fireproofing.”
“Use of asbestos millboard and asbestos cement to fit transom panels beneath windows at the Guardian / AXA insurance building. Assuming an equal share of tax year 1966/67 as one of four employers, and one day per week working with asbestos products as opposed to his main shuttering work, exposure duration approximately equivalent to 0.05 working years. Assuming a 50:50 split between cutting/handling asbestos millboard at an average of 5 f/ml and asbestos cement at an average 0.1 f/ml. As work in 1967/68 was one of six employers there may be a small additional exposure for any work done in the few weeks worked in that year. Estimated dose contribution 0.13 f/ml years.”
“In 1996/67 I worked for Bovis limited Lytham. I helped in the construction of the Guardian Insurance building in Blackpool. I think that this is now the AXA building. Again, my job was to make shuttering and therefore I was not exposed to asbestos dust when shuttering. However, I used millboard at times. I recall cutting it with a handsaw and installing it under windows sills when working on the Guardian Insurance building. Millboard is a brownie colour. Asbestos is lighter grey. The asbestos would be used first and then the millboard next. Finally, a decorative finish would be applied.”
“If there are any differences between applications that I have made to the DWP and Laura Martin’s report I confirm that it is Laura Martin’s report that is accurate. I say this because I've had more time to consider my employment history and asbestos exposure when meeting Laura that I had at the time that I applied to the DWP.”
“He described fitting panels beneath the windows, with asbestos cement on the outer side and asbestos millboard on the inner side as an insulative layer. I showed Mr Harrison pictures of asbestos millboard and a similar non asbestos fibreboard without pre identifying which was which to him. Mr Harrison indicated the asbestos version as being the product he had used on this project.”
“Mr Harrison was able to distinguish between photographs of asbestos millboard and flat asbestos cement sheeting without any prompting and identify them to me as the products he used on that particular project.”
“Very flaky. A lot of dust came off. The other was harder to cut.”
“Hard but could be cut with a saw.”
“No it wouldn’t be that. It wouldn’t be that. It wouldn’t be that. I just can’t understand that.”
“She showed me lots of examples and I recognised a lot of examples of what was, you know, dangerous asbestos products.”
“I had no more information than the first statement. I would have recorded any new information.”
“With the passage of time I am not sure, it is firming up my recollections of conversations that are not in my notes and clearing up the aspects that were written up about the Guardian Building and work he did for Bovis.”
“I cannot give a definitive answer. In the first report it is not clear enough that the AXA building work described was work with Bovis. I can’t recall why.”
“asbestos sheeting is what Mr Harrison meant by the asbestos.”
“I am confident they are the ones he saw.”
“He told me personally that it was asbestos. He did identify the asbestos containing version from the photos. I did not put that in the first report’ and ‘he recalled clearly and could distinguish between them. My recollection was he could remember and he used asbestos millboard.”
“In conclusion as stated I do consider it plausible that the insulating material/board of some kind was fitted under windows, behind the asbestos cement. I was drawn to a non-asbestos board by the comment ‘browney’ but overall, I consider the various sources of evidence too uncertain to draw conclusions as to whether, on the balance of probabilities, the millboard was in asbestos or non-asbestos containing board.”
“the asbestos would be used first and then the millboard next. Finally, a decorative finish would be applied.” (v) In addition, the first colour described for the millboard was “browney”
“We agree that should the court accept that the deceased did work with AC the most likely fibre type would have been chrysotile asbestos.”
“Before 1965 neither the industry generally nor those responsible for safety and health nor the factory inspectorate nor the medical profession suggested that it was necessary or even that it would be prudent for the risks arising from familial exposure to be addressed by the industry. In truth the alarm did not sound until late 1965 when it began to be appreciated that there could be no safe or permissible level of exposure direct or indirect to asbestos dust. There after the learning curve about the risk arising from familial exposure was fairly steep.’”
“The use and manipulation of asbestos and asbestos cement products is very diverse and ranges from the fixing of occasional single sheets to extensive operations involving cutting or machining by power driven equipment. While care should always be exercised special precautions are only necessary when there is a possibility that operatives may inhale asbestos dust as result of proximity to cutting grinding or similar operations.”
“3.2.1 where hand cutting and working has to take place regularly a dust exhaust system will often not be possible. Where any risk of inhaling asbestos dust is present operatives should wear approved type respirators.”
“In my view Mr Harrison's short-term high-level exposures would certainly have exceeded the fibre levels as described above. Some of the work with asbestos insulating board could have been below at times and with asbestos cement consistently below.”
“I am not persuaded that this exposure albeit noting the uncertainties, carried a foreseeable risk of injury. Whilst it seems it occurred after the seminal 1965 work of Newhouse and Thompson, the ARC when they produced guidance in April 1967 were advising little or no precautions for small scale intermittent work where hand tools were used, albeit they did for all work, advised dust free methods of cleaning. Whether the second defendant were aware or should have been aware of this guidance is a matter for the court.”
“‘The first question is whether Anglia should at any time during Mr Bussey's employment - that is, between 1965 and 1968 (the precise dates are not known) - have been aware that the exposure to asbestos dust which his work involved gave rise to a significant risk of asbestos-related injury. (I say "significant" only so as to exclude risks which are purely fanciful: any real risk, albeit statistically small, of a fatal illness is significant.) That will depend on how quickly the knowledge, first widely published in 1965, of the fact that much lower exposures than had previously been thought to be dangerous could cause mesothelioma was disseminated among reasonable and prudent employers whose employees had to work with asbestos. One aspect of this question is whether, even though Anglia may have been aware of the risk in general terms, it was reasonable for it at the material time to believe that there was a level of exposure below which there was no significant risk, and that Mr Bussey's exposure was below that level.”
“At the time Anglia had no way of measuring the actual level of asbestos to which Mr Bussey was exposed……. all that Anglia knew, or ought to have known, was that Mr Bussey’s work could regularly exposed him to small quantities of asbestos dust’. Later he said [57] ‘As things stood in 1965-1968 Anglia could not know one way or the other whether the extent of Mr Bussey’s exposure was liable to cause mesothelioma. It might or it might not do so. There were ready means of reducing that risk. In my view if the judge had not felt constrained by the decision in Williams v University of Birmingham [2012] PIQR P4 he might have concluded that as a reasonably prudent employer Anglia ought to have foreseen that risk; since that risk could be avoided by simple precautions, it was not a risk which ought to be accepted.”
“From about the 1960s, it became widely known that exposure to asbestos dust and fibres could give rise not only to asbestosis and other pulmonary diseases, but also to the risk of developing a mesothelioma. This is a malignant tumour, usually of the pleura, sometimes of the peritoneum. In the absence of occupational exposure to asbestos dust it is a very rare tumour indeed, afflicting no more than about one person in a million per year. But the incidence of the tumour among those occupationally exposed to asbestos dust is about 1,000 times greater than in the general population, and there are some 1,500 cases reported annually. It is a condition which may be latent for many years, usually for 30-40 years or more; development of the condition may take as short a period as 10 years, but it is thought that that is the period which elapses between the mutation of the first cell and the manifestation of symptoms of the condition. It is invariably fatal, and death usually occurs within 1-2 years of the condition being diagnosed. The mechanism by which a normal mesothelial cell is transformed into a mesothelioma cell is not known. It is believed by the best medical opinion to involve a multi-stage process, in which 6 or 7 genetic changes occur in a normal cell to render it malignant. Asbestos acts in at least one of those stages and may (but this is uncertain) act in more than one. It is not known what level of exposure to asbestos dust and fibre can be tolerated without significant risk of developing a mesothelioma, but it is known that those living in urban environments (although without occupational exposure) inhale large numbers of asbestos fibres without developing a mesothelioma. It is accepted that the risk of developing a mesothelioma increases in proportion to the quantity of asbestos dust and fibres inhaled: the greater the quantity of dust and fibre inhaled, the greater the risk. But the condition may be caused by a single fibre, or a few fibres, or many fibres: medical opinion holds none of these possibilities to be more probable than any other, and the condition once caused is not aggravated by further exposure. So if C is employed successively by A and B and is exposed to asbestos dust and fibres during each employment and develops a mesothelioma, the very strong probability is that this will have been caused by inhalation of asbestos dust containing fibres. But C could have inhaled a single fibre giving rise to his condition during employment by A, in which case his exposure by B will have had no effect on his condition; or he could have inhaled a single fibre giving rise to his condition during his employment by B, in which case his exposure by A will have had no effect on his condition; or he could have inhaled fibres during his employment by A and B which together gave rise to his condition; but medical science cannot support the suggestion that any of these possibilities is to be regarded as more probable than any other. There is no way of identifying, even on a balance of probabilities, the source of the fibre or fibres which initiated the genetic process which culminated in the malignant tumour. It is on this rock of uncertainty, reflecting the point to which medical science has so far advanced, that the three claims were rejected by the Court of Appeal and by two of the three trial judges.”
“To the question posed in paragraph 2 of this opinion I would answer that where conditions (1)-(6) are satisfied C is entitled to recover against both A and B. That conclusion is in my opinion consistent with principle, and also with authority (properly understood). Where those conditions are satisfied, it seems to me just and in accordance with common sense to treat the conduct of A and B in exposing C to a risk to which he should not have been exposed as making a material contribution to the contracting by C of a condition against which it was the duty of A and B to protect him. I consider that this conclusion is fortified by the wider jurisprudence reviewed above. Policy considerations weigh in favour of such a conclusion. It is a conclusion which follows even if either A or B is not before the court. It was not suggested in argument that C's entitlement against either A or B should be for any sum less than the full compensation to which C is entitled, although A and B could of course seek contribution against each other or any other employer liable in respect of the same damage in the ordinary way. No argument on apportionment was addressed to the House. I would in conclusion emphasise that my opinion is directed to cases in which each of the conditions specified in (1) - (6) of paragraph 2 above is satisfied and to no other case.”
“Section 3(1) Does not state that the responsible person will be liable in tort if he has materially increased the risk of a victim of mesothelioma. It states that the section applies where the responsible person is liable in tort for materially increasing that risk. Whether and in what circumstances liability in tort attaches to one who has materially increased the risk of a victim contracting mesothelioma remains a question of the common law.”
“What constitutes a material increase in risk? ”
“For the claim to succeed, the judge needed to be satisfied that the extent and duration of the exposure had constituted a material increase in the risk to the Deceased of contracting mesothelioma. No specific measurement of the duration is necessary and the Recorder was right to resist the invitation to fix one. Exposure that would fall within the de minimis formula would be insufficient. However, the type of contract work undertaken by International Combustion at power stations and the role of the Deceased in that work, coupled with his generic description of conditions in power stations at the time, undoubtedly justified the finding that this was not a de minimis case. Mr Limb frankly conceded that to work in such conditions at a particular location for a week would not be de minimis.”
“Thus the conundrum is answered by saying that there are special features about mesothelioma, and the gaps in our knowledge in relation to it, that render it inappropriate to decide causation on epidemiological data as to exposure. So far as concerns apportionment between tortfeasors jointly liable for causing mesothelioma it is likely to be necessary to use epidemiological evidence faute de mieux.”