"not all the horses are mine: some are Jamie"s"
"the smell was disgusting. I was quite upset by the whole thing."
"the quantity of feed was disproportionate to the number of feeders" and "there was a distinct lack of food available to the horses."
"the physical scale of effort required for basic care was well beyond the people and the facilities there. A small army of people were required over the next few weeks to look after their wider needs."
"we provide basic standards of care for animals. The luxury is having vets on site. I am not imposing an unreasonable standard. We educate people to what are easily attainable standards of horse care. If you bring an animal into ownership or responsibility and if it is debilitated, you get a vet to find out what is the problem both for the sake of that animal and for the sake of the collection."
"emaciated" means that there is no covering fat or muscle and the prominences are angular; "very thin" means that there is a little fat maybe on the pelvis and neck. For example, she said that Mr Parker's "scores" for RS83 was "not reasonable"; RS89 "it was thinner than Mr Parker's "score" of 3/10; RS91 was "closer than the others"; RS66 and RS91 were "not underscored by Veterinary Surgeon De Brauwere;"
"bots" are insects that lay eggs on the horse's hair which hatch into maggots when the horse eats them. They burrow into the tongue and end up in the stomach. Ascarids infect foals and young horses. In such animals an absence would indicate that, at some stage, they had received worming treatment. Most older horses develop an immunity. The other parasites mentioned were tapeworms. "
"there is a marked leucocytosis. There is moderate hypoalbuminaemia. Total protein, total globulin, alpha2 and beta1 globulin fractions are markedly raised. CK [Creatinine kinease: page 24 of the BCL booklet] is slightly raised but all other figures are within normal limits. These results suggest the response to an infectious/inflammatory process with tissue damage and protein losing enteropathy related to significant endoparasitism. There is no evidence to suggest that an underlying metabolic abnormality is involved."
"Diagnosis of the cause of poor condition in equine welfare cases." "
"cases of starvation/neglect often have multi-factorial problems relating to malnutrition, over crowding, poor dentition, endo- and ecto- parasitism, lack of shelter, stress etc. Therefore interpretation of white blood cells is not straightforward as other factors such as infection [usually causing an increased white cell count] are involved."
"the diagnosis of starvation cannot be made on the basis of blood sampling. Blood samples can rule out some important causes of emaciation or low body score, but I would emphasise the importance of any veterinary surgeon considering the full clinical picture. The visual appearance and demeanour of the horse speaks leagues. Consideration of access to adequate food and water, provision of adequate shelter, appropriate stocking density, effective worm control and medical care are all part of proper horse care. Blood sample results cannot provide evidence of whether a horse is starved or well fed or reliably assess where the horse is on the wide spectrum between the two."
"Mr Gray showed me round the Farm buildings. He explained his management and how he conducted his business. He does all the work himself with the help of his family of wife, son and two daughters. If they are all away together on holiday, a competent horseman moves in to manage the yard."
"Fuck off; you"re not cautioning me."
"I think that I am acting on behalf of all the Appellants."
"he does all the work with the help of his family of wife, son and two daughters."
"as far as possible [I] subjected the horses there to a general health examination to provide a short report on the current state of each animal."
" meaning moribund with an expectation of death within a couple of hours; a collapsed animal. 10 would be grossly fat. 3 is borderline: with varying levels of concern depending on the age of the animal: younger less concerning than older. 111 We were taken through his written assessments. It is clear that John Parker did not particularly recollect the individual horses. He looked at the records and was invited to consider the photographs. When he did so, he was prepared to amend his score. We were not impressed by this method of scoring and consequently there were significant differences between his scores and those from other Veterinary Surgeons. Sadly the plain truth is that theirs were much more thorough and careful than his and we are entirely satisfied that we should prefer theirs. We do appreciate the difficulties in evaluating body scores. Individuals have used different scales - either the one which that person was familiar with or the one with which they were asked to work. In order to test the quality it is the thoroughness of the examination that matters. Veterinary Surgeon De Brauwere took 40 to 45 minutes per animal assisted b Ms Kirton; John Parker ½ a minute. John Parker's notes are cursory as were, we find, the examinations themselves. The scores that he offers simply do not measure up in qualitative terms to those of the others. Understandably he did not recall individual animals at all. He did make a valid criticism: these scoring methods are not designed for dead animals. But that information does offer us assistance in deciding upon the information proffered on the dead animals as to the state of them as described by Veterinary Surgeon Hayes. His was not a proper post mortem as we have seen, but John Parker still acknowledged Veterinary Surgeon Hayes" professionalism as do we. Veterinary Surgeon Hayes offered us guidance and we are entirely satisfied that we should accept it. John Parker started with the horses at Moat Farm. Some were not specifically identified; others RS8, RS10, RS12-17, RS26-29, RS31, RS33-50, RS67, RS69 and RS71-72 were noted and identified. 3 of these - RS27, RS47 and RS48 had been claimed by the Fourth Appellant. Most of these were spoken of generally rather than specifically by Mrs Ringer and Mrs Verhulst. Mrs Verhulst gave evidence in detail about only RS28, RS72 and the horse that subsequently died: RS37. Peter Green dealt with this equine in more detail. There was no cross examination about the others. Then he went to ILPH and inspected RS53-61. These are animals inspected by Mr Willamson and we have considered John Parker's comments alongside his. John Parker acknowledged that he did not recall these animals "well enough."
"maybe I did not answer the question last time particularly well."
"losses of up to 10% are acceptable in livestock farming and would be acceptable at low level horse dealing/farming. Losses of 10% or more within a six week period are not acceptable and, if they occur, veterinary attention should be sought."
"Usually a horse affected by cyathostomiasis is, or is becoming thin, and will often have ventral oedema and altered faecal texture ranging from "cowpat" diarrhoea to profuse watery material. Intermittent fever, occasional colic, and a normal to ravenous appetite are sometimes seen. Late in the disease, however, the animal may become anorectic. Coat condition and quality is often poor. However it is possible and certainly within my experience that horses collapse from cyathostomiasis without showing any sign of diarrhoea, very poor body condition or any particularly obvious clinical harbinger. If there is encysted worm burden the rupture of millions of larvae is sufficient to cause an acute shock syndrome and sudden death and these bright red larvae can be seen in the faeces."
"epg" [eggs per gram] and "eggs seen" [Mc Masters]. She purported to justify the table. However before District Judge Vickers, she had accepted that it was misleading and had apologised. To us she said that it was a slight error and stood by her evidence. We reject it. After the short adjournment, issues arose about the cross examination that had been put to Peter Green and Ms Barralet and the evidence that Madeleine Forsyth was giving. The Judge stopped cross examination where it went beyond that scope of what had previously been put. Initially an attempt was made by Madeleine Forsyth and, to an extent by Ms Howe, to assert that matters being adduced had been put to both of them. A short study of the transcript of Ms Barralet's evidence revealed the contrary. The Judge said that, if the matters which were being pursued were to continue he would have to consider whether either or both Peter Green or Ms Barralet would have to be recalled if that was the Respondents" application. When examination in chief was completed, time was given to Mr Fullerton to decide what he wished to do. In the event he decided not to pursue his application to adduce challenged evidence and the preliminary arrangements for the Court to convene to hear submissions were abandoned. The remainder of Ms Forsyth's evidence was confined largely to her confirmation of the remaining parts of her report, from [1652] onwards. She confirmed that her opinion was that the death and condition of the horses was "a consequence of various factors" [554] and that she supported parasitism over malnutrition. It was not safe to make a diagnosis of emaciation since the suggested protocol outlined by Peter Green in his article had not been carried out and, indeed, some equines still showed PLE after 10 days from seizure. She confirmed that both parasitism and enteric gut infection [salmonellosis] are treatable conditions depending upon the extent of them. Salmonellosis was a likely cause of death. So what about cyathostomiasis? She replied that it was involved but she did not know what killed the horses. She adhered to her remarks in paragraph 1-4 of the addendum report at [1623] and to her summary. 125 Essentially her thesis is that the causes of death and disease were multi factorial and she was unable to say which predominated. She did not know and could not be sure if the carcasses had died by acute larval cyathostomiasis but she would have expected some signs before death. She conceded that she had agreed [557 question 20] that a poor horse would not have died in less than two days. She appeared to us to qualify it by saying that she had not carefully considered it. Then she accepted that a responsible carer would have noticed signs in two days. In her final report [561] she advanced Type I cyathostomiasis. She conceded that it does not cause death generally. Then she qualified that by saying that, depending upon the state of the condition and the length of time that it had existed, an animal could be so damaged that it was vulnerable to a secondary condition. But with Type I cyathostomiasis the symptoms would be noticeable to a caring owner; that caring owner would worm the animal, feed it properly and, if necessary, call a Veterinary Surgeon. If the horse was in poor condition there was no excuse: the owner should focus on feed and environment and call a Veterinary Surgeon if the issues were not resolved. There was no excuse for the Shetlands and the donkeys being in poor condition. In the case of the carcasses she accepted that there were no tell tale signs of Type II cyathostomiasis: no red larvae, no cobblestone or poppy seed effect of the gut. So her theses have been: salmonellosis, Type II cyathostomiasis and Type I cyathostomiasis. She claimed that her views had remained unchanged and we looked at clarified passages in her reports. In our judgement she has shifted her ground. Both she and John Parker have remained loyal to the Appellants to the end. Their evidence cannot begin to compete with the vast array of other better argued and better researched evidence that we have heard. To us they have both attempted to open new avenues of exculpation as old ones ceased to be tenable. They lost their objectivity and impartiality in the process. We are entirely satisfied that we must reject their efforts and we do so. 126 CHARGES We have considered the written submissions. We consider that we have dealt with most of them in our findings and consideration of the evidence. Charge 1: AGAINST the First Appellant AND the Fifth Appellant This charge alleges causing unnecessary suffering to the carcasses by failing to exercise reasonable care and supervision in respect of protection. This is dealt with in the joint report at [551]. None of these animals were humanely destroyed. The experts are agreed that it is extremely unlikely that they all died without many of them showing signs of illness or collapse prior to death. Such a large number of deaths in such a space of time suggests a common causative factor that should have been investigated. Having eliminated other possible causes it is plain that they were left to die. No Veterinary Surgeon was called. C20 clearly needed a Veterinary Surgeon but equally clearly did not get the attention of one. It too was left to die in the trailer into which it had been unceremoniously hauled in Hughenden. Allowing horses to die without care, veterinary attention or euthanasia, breaks all acceptable standards. They were left where they fell or removed to a place where they could be left to rot. We are satisfied that these horses were neglected and starved. It follows that the Appellants failed to exercise reasonable care and supervision in respect of protection and that they knew or ought to reasonably to have known that this would have that effect. Charge 2: AGAINST ALL APPELLANTS This Charge alleges that the needs of the carcasses were not met to the extent required by good practice in that their needs to be protected from pain, injury, suffering and disease were not met. This is dealt with in the joint report alongside Charge 1. Much the same as was said above applies here too. Allowing horses to die does not meet their needs to be protected from pain, injury, suffering and disease. We are satisfied that this is what happened. We have found that all the Appellants were responsible for all these animals and that they knew what was happening on the premises and took no action to alleviate the problems. Charge 3: AGAINST the First Appellant AND the Fifth Appellant KH1 and the allegation that the Appellants caused unnecessary suffering in that they failed to provide this animal with a nutritionally balanced diet, including water. Its last act was to drink copiously water from a bucket. It was then euthanased. The experts [551] are agreed that this horse was collapsed and emaciated. It was suffering and humane disposal was inevitable. It was terminally ill from a number of factors. 127 At the time of discovery, the First Appellant had been leaving the Yard to collect a part for his non functioning skid loader. The animal was unable to stand. It had chronically injured itself by rubbing flesh from its side whilst trying to alleviate its condition. It had been down for a significant period -"several hours" as the Veterinary Surgeon agree. We accept that this horse was suffering from malnutrition, parasitism and terminal salmonellosis. We are satisfied that this animal suffered unnecessarily by being neglected and starved. It could not get to food or water and no attempt was made to provide it with either. Both the First and the Fifth Appellants either knew or ought to have known of the condition of this animal and took no action. Charge 4: AGAINST the First Appellant AND the Fifth Appellant The KH horses. KH2, KH4, KH5. KH6, KH7, KH8, KH9, KH10, KH16, KH17, KH18, KH19 and KH20. The allegation here is that the unnecessary suffering was caused by failing to provide them with a suitable diet. The experts disagree on this charge except that they do concur that KH16 and KH17 were unacceptably thin. The evidence from Veterinary Surgeon Robinson was to the effect that they were emaciated or very thin. We accept that evidence. Veterinary Surgeon Jepson also described them in his evidence as "appallingly thin."