“For the last year, difficult time” “Feel exhausted and concentration, not sleeping” “Stressful job … senior IT consultant” “Pressure to perform”
“Now in a good amount of pain, constant discomfort”
“Can we please explore the possibility of an EOW (Expression of Wish) for me due to my current inability to fully perform my work tasks as a result of medical issues resulting from a workplace accident. I would like to understand my options sooner rather than later.”
“Peter has not been engaged for a sufficient time with any one client to influence either their innovation, agenda or service excellence scores.”
“These viscoelastic effects of rapid deformation prompt a classification of dynamic injuries where the applied forces occur in less than 50 milliseconds. Thus, axonal injury is dependent on both the magnitude of strain and the rate of strain during brain trauma… this mechanism may potentially explain the large extent of midline damage that has been historically noted in human DAI including damage found in the corpus callosum. The forces required to cause the tissue deformation inducing DAI are often misunderstood, a common cause of confusion is the type of forces required to induce axonal injury because DAI has been observed in cases of falls and assaults as well as in victims of motor vehicle accidents … inertial forces such as those produced by rotational acceleration of the head during automobile crashes often culminate in the contact force of the head striking the interior of the automobile. Thus, although the formation of DAI is produced by inertial forces, contact forces often cause the levels of acceleration necessary to produce DAI”
“Although conventional brain imaging techniques are useful for revealing macroscopic changes in severe DAI such as white matter tears and parenchymal haemorrhage, these techniques cannot easily detect the predominant pathology microscopic axonal swellings. Accordingly, patients and animal models with little macroscopic injury following diffuse brain injury may have normal appearing images of brain, leading many to believe that axonal pathology is substantially under diagnosed.”
“The above difficulties are individually consistent with either organic impairment or psychological disturbance or with both. Acquired anxiety and low mood may produce some of the difficulties and aggravate difficulties of organic origin. Difficulties with memory and concentration and with heightened fatigue have been reported by Mr Siegel prior to the accident and in conjunction with both physical and psychological problems. Where cognitive difficulties and heightened fatigue are reported in the absence of feeling subjectively distressed or agitated, the prime source of these would appear to be organic impairment associated with cerebral injury. His sudden personality changes, including ballistic temper outbursts, such as have been shown by Mr Siegel, are also typical of cerebral injury, however, heightened irritability may be aggravated by symptoms of anxiety disorder, by anxious ruminations, by low mood, and by the frustrations associated with cognitive defects. A vicious circle is then created in that such difficulties may lead to lowered mood, agitation and frustration which may further reduce performance, self-confidence and motivation.”
“Mechanism of injury, clinical history and the presence of post-traumatic amnesia for several days indicate that he suffered a severe head injury with associated cerebral injury. He also shows momentary retrograde amnesia. At assessment, one year following the accident, he continued to suffer cognitive difficulties consistent with both cerebral injury and, to a lesser degree, with psychopathology. These include deficits in: memory, concentration, organising and planning, following group conversations, mental arithmetic, reading, decision making, multi-tasking, abstract thinking, and social judgment. During the course of assessment he showed difficulties in focusing on relevant information, his speech was garrulous and intense in often unnecessary detail. He had difficulties in keeping to the point, despite being aware of the limits to the time available as it grew late in the evening. Such cognitive difficulty, together with persisting heightened fatigue, lowered tolerance and reduced verbal control have had a debilitating effect on his workplace skills and interactions with colleagues and clients. He reported persisting headaches, sleep disturbance and reduced tolerance for alcohol. He shows a pre-accident history of migraine headaches, sleep disturbance and heightened fatigue. Indications are that headaches have increased in frequency since the index accident. He shows personality changes including impulse behaviour, reduced motivation, and indifference to others. He also showed heightened irritability and increased frequency and severity of temper outbursts which have had a detrimental effect on his interpersonal relationships. Psychologically he has developed agoraphobis with panic attacks and symptoms of partial PTSD.”
“There is a lack of evidence that Mr Siegel sustained a head injury, there is a lack of evidence that he sustained a brain injury, there is a lack of evidence that he had, as a consequence of his accident diffuse axonal injury. There is a lack of evidence that he had significant neuropsychological abnormalities on clinical testing. The evidence that he has sustained cerebral injury appears to have arisen from the late Mr Price (a neurosurgeon who has … no expertise in psychiatric disturbances) and Ms Levett who has not only been shown to be unreliable but has been removed from her professional practice because of her unprofessional and biased activities. The new evidence appears to come from Dr Allder, but I am at a loss to understand the neurological explanations that he put forward. I am unaware of his psychiatric expertise or training.”
“it is not clear … how the Claimant could recall for me events that he has no memory of. If he has no memory of them then it would not be possible for him to relate them to me. The important point is that he has a pattern of recall following the material accident which does not follow the pattern of that which occurs following neurological injury.”
“There are many symptoms that he has which are not typical of brain damage. First … post-traumatic amnesia … secondly … the onset of tunnel vision, thirdly … light sensitivity, fourthly … emotional numbness, fifthly, … the pattern of his headache, sixthly … loss of topographical orientation, seventhly … the pattern of the development of symptoms over time which do not compose a syndrome typical of somebody with brain damage.”
“He shows very superior intelligence with strength in processing speed. He has a mildly but significantly reduced memory, particularly from verbal material. His executive functioning is consistent with his intellectual functioning.”
“I have also conducted my own thorough review of the matter … and concluded that the only appropriate sanction in all the circumstances of this case is that the registrant should be struck off. In my judgment, the persistent breach of professional boundaries by the registrant was so serious and a lack of insight into the inappropriateness of her own conduct so profound that striking off is the only appropriate and proportionate sanction which both reflects the gravamen of the case and the protects the public interest.”