“There is evidence of hypoxic/ischaemic damage to the brain and spinal cord. This indicates that there has been a significant interruption of the oxygen (hypoxic) and blood (ischaemic) supply. This is demonstrated by the presence of red neurons and a vascular pattern of axonal injury. …. I have specifically considered the possibility of traumatic brain injury. While hypoxia/ischaemia may be one of the consequences of brain trauma, it is not specific, and I have not found specific evidence in the brain to indicate trauma. There are features (notably the cerebral haemorrhage and the axonal injury in the cervical spinal cord) where I have considered the possibility of trauma, but in my opinion, these are more likely to be the complications of hypoxia/ischaemia, based on their morphological pattern.”
“In fatal cases of non-accidental/abusive head trauma, the typical constellation of features includes intracranial subdural haemorrhage and encephalopathy, with retinal haemorrhages present in a proportion of cases. While it is true that bilateral, extensive, multi-layered retinal haemorrhages extending to the peripheral retina are considered quite specific for abusive head trauma, in P’s case, intracranial subdural haemorrhage was not seen at autopsy examination or on neuropathological examination, and I found no significant optic nerve sheath haemorrhage. Professor Jacques did not find specific evidence in the brain to indicate trauma, and in his opinion, the cerebral haemorrhage and axonal injury in the cervical spinal cord are more likely to be complications of hypoxia/ischaemia. Therefore, the overall features in this case are not in keeping with severe head trauma. …. Although I await Dr Cary’s full autopsy report, from the history given, from information provided this appears to be a case of a complex positional asphyxia, with P prone, with head below the body, under the radiator and between the mattress and the wall…. …. …. this is a highly unusual case of bilateral, extensive, multilayered retinal haemorrhages extending to the peripheral retina, with accompanying brain swelling and hypoxic-ischaemia, but without other features of abusive head trauma such as intracranial subdural haemorrhage or optic nerve sheath haemorrhage. It may be that a number of contributory factors acted together in this case to cause unusually extensive retinal bleeding, where each alone would not typically be considered a plausible sole cause.”
“As you are aware, I am unable to complete my final report due to outstanding results. However, to assist the Court I am able to state the following having considered the external and internal postmortem findings, the neuropathology, the ophthalmic pathology and the interim opinion of Dr Cartlidge. 1 Essentially, I agree with opinions of Dr Cartlidge concerning the circumstances leading up to death. 2 There were no external or internal marks of injury and a skeletal survey was negative for fractures. 3 The principal finding in the brain was one of hypoxiaischaemia. 4 The main findings in the eyes were retinal haemorrhages. Importantly there was no evidence of optic nerve sheath haemorrhage commonly seen in cases of head injury. 5 At this stage the main finding is one of asphyxia, the cause of which is not apparent from the postmortem findings alone. 6 This is the sort of case where any final conclusions are heavily dependent on the circumstantial evidence. 7 In this case there is potential evidence of positional asphyxia with an element of wedging, as well as suffocation from bedding. In relation to these possibilities I accept the careful reasoning of Dr McPartland in relation to the origin of the retinal haemorrhages. 8 Toxicological results are still outstanding so I cannot absolutely exclude some toxicological contribution. 9 The final exclusion of head injury will await the examination of the neck by Professor Mangham as there can be subtle changes that imply an element of flexion / extension of the kind seen in shaking injury. However even on the basis of the information available shaking seems unlikely in the absence of both thin-film subdural haemorrhages and optic nerve sheath haemorrhages.”
“19.As identified by Hedley J in the Redbridge case, applications to withdraw care proceedings will fall into two categories. In the first, the local authority will be unable to satisfy the threshold criteria for making a care or supervision order under s.31(2) of the Act. In such cases, the application must succeed. But for cases to fall into this first category, the inability to satisfy the criteria must, in the words of Cobb J in Re J, A, M and X (Children), be “obvious”.” 20.In the second category, there will be cases where on the evidence it is possible for the local authority to satisfy the threshold criteria. In those circumstances, an application to withdraw the proceedings must be determined by considering (1) whether withdrawal of the care proceedings will promote or conflict with the welfare of the child concerned, and (2) the overriding objective under the Family Procedure Rules. The relevant factors will include those identified by McFarlane J in A County Council v DP which, having regard to the paramountcy of the child’s welfare and the overriding objective in the FPR, can be restated in these terms: (a) the necessity of the investigation and the relevance of the potential result to the future care plans for the child; (b) the obligation to deal with cases justly; (c) whether the hearing would be proportionate to the nature, importance and complexity of the issues; (d) the prospects of a fair trial of the issues and the impact of any fact-finding process on other parties; (e) the time the investigation would take and the likely cost to public funds.”